Inhibition of nuclear hormone receptor activity by calreticulin.
We have shown that a polypeptide of M(r) 60,000 (60K) that shares N-terminal homology with a calcium-binding protein, calreticulin, can bind to an amino-acid sequence motif, KXGFFKR, found in the cytoplasmic domains of all integrin alpha-subunits. The homologous amino-acid sequence, KXFFKR (where X is either G, A or V), is ... also present in the DNA-binding domain of all known members of the steroid hormone receptor family; amino acids in this sequence make direct contact with nucleotides in their DNA-responsive elements and are crucial for DNA binding. Here we show that both the 60K protein (p60), purified on a KLGFFKR-Sepharose affinity matrix, and recombinant calreticulin can inhibit the binding of androgen receptor to its hormone-responsive DNA element in a KXFFKR-sequence-specific manner. Calreticulin can also inhibit androgen receptor and retinoic acid receptor transcriptional activities in vivo, as well as retinoic acid-induced neuronal differentiation. Our results indicate that calreticulin can act as an important modulator of the regulation of gene transcription by nuclear hormone receptors.
Mesh Terms:
Amino Acid Sequence, Animals, Base Sequence, Calcium-Binding Proteins, Calreticulin, Cell Line, Cell Nucleus, DNA, Gene Expression Regulation, Integrins, Molecular Sequence Data, Rats, Receptors, Androgen, Receptors, Retinoic Acid, Ribonucleoproteins, Tumor Cells, Cultured, Vero Cells
Amino Acid Sequence, Animals, Base Sequence, Calcium-Binding Proteins, Calreticulin, Cell Line, Cell Nucleus, DNA, Gene Expression Regulation, Integrins, Molecular Sequence Data, Rats, Receptors, Androgen, Receptors, Retinoic Acid, Ribonucleoproteins, Tumor Cells, Cultured, Vero Cells
Nature
Date: Feb. 03, 1994
PubMed ID: 8107809
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