Structural characterization of a novel Cbl phosphotyrosine recognition motif in the APS family of adapter proteins.

The Cbl adapter proteins typically function to down-regulate activated protein tyrosine kinases and other signaling proteins by coupling them to the ubiquitination machinery for degradation by the proteasome. Cbl proteins bind to specific tyrosine-phosphorylated sequences in target proteins via the tyrosine kinase-binding (TKB) domain, which comprises a four-helix bundle, an ...
EF-hand calcium-binding domain, and a non-conventional Src homology-2 domain. The previously derived consensus sequence for phosphotyrosine recognition by the Cbl TKB domain is NXpY(S/T)XXP (X denotes lesser residue preference), wherein specificity is conferred primarily by residues C-terminal to the phosphotyrosine. Cbl is recruited to and phosphorylated by the insulin receptor in adipose cells through the adapter protein APS. APS is phosphorylated by the insulin receptor on a C-terminal tyrosine residue, which then serves as a binding site for the Cbl TKB domain. Using x-ray crystallography, site-directed mutagenesis, and calorimetric studies, we have characterized the interaction between the Cbl TKB domain and the Cbl recruitment site in APS, which contains a sequence motif, RA(V/I)XNQpY(S/T), that is conserved in the related adapter proteins SH2-B and Lnk. These studies reveal a novel mode of phosphopeptide interaction with the Cbl TKB domain, in which N-terminal residues distal to the phosphotyrosine directly contact residues of the four-helix bundle of the TKB domain.
Mesh Terms:
Adaptor Proteins, Vesicular Transport, Adipose Tissue, Amino Acid Motifs, Amino Acid Sequence, Animals, Binding Sites, CHO Cells, Calcium, Calorimetry, Cricetinae, Crystallography, X-Ray, Electrons, Immunoblotting, Immunoprecipitation, Kinetics, Models, Molecular, Molecular Sequence Data, Mutagenesis, Site-Directed, Phosphorylation, Phosphotyrosine, Protein Binding, Protein Conformation, Protein Structure, Tertiary, Signal Transduction, Thermodynamics, Transfection, Tyrosine
J. Biol. Chem.
Date: May. 13, 2005
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