Opposing classes of prp8 alleles modulate the transition between the catalytic steps of pre-mRNA splicing.
The spliceosome is thought to undergo a conformational change between the two catalytic steps of precursor messenger RNA splicing, although the specific events in this transition are poorly understood. We previously proposed a two-state model of splicing in which the conformations required for the first and second steps are in ... competition. Here, we identify and characterize a class of prp8 mutants that suppress first-step splicing defects and oppose the action of the previously described prp8 suppressors of second-step defects; these opposing effects parallel those of ribosomal 'ram' and 'restrictive' mutants, which alter fidelity of transfer RNA decoding. On the basis of genetic interactions, we propose that prp8-mediated substrate repositioning during the transition occurs between catalytic-center opening and closure mediated by the U6 small nuclear RNA and the DExH/D ATPase gene prp16. Modulation of these events alters splice-site selection and splicing fidelity.
Mesh Terms:
Adenosine Triphosphatases, Alleles, DNA Primers, Models, Molecular, Mutation, Protein Conformation, RNA Helicases, RNA Splicing, RNA, Small Nuclear, Ribonucleoprotein, U4-U6 Small Nuclear, Ribonucleoprotein, U5 Small Nuclear, Saccharomyces cerevisiae, Saccharomyces cerevisiae Proteins, Spliceosomes
Adenosine Triphosphatases, Alleles, DNA Primers, Models, Molecular, Mutation, Protein Conformation, RNA Helicases, RNA Splicing, RNA, Small Nuclear, Ribonucleoprotein, U4-U6 Small Nuclear, Ribonucleoprotein, U5 Small Nuclear, Saccharomyces cerevisiae, Saccharomyces cerevisiae Proteins, Spliceosomes
Nat. Struct. Mol. Biol.
Date: Jun. 01, 2007
PubMed ID: 17486100
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