CDK11p58 represses vitamin D receptor-mediated transcriptional activation through promoting its ubiquitin-proteasome degradation.
Vitamin D receptor (VDR) is a member of the nuclear receptor superfamily and regulates transcription of target genes. In this study, we identified CDK11(p58) as a novel protein involved in the regulation of VDR. CDK11(p58), a member of the large family of p34cdc2-related kinases, is associated with cell cycle progression, ... tumorigenesis, and apoptotic signaling. Our study demonstrated that CDK11(p58) interacted with VDR and repressed VDR-dependent transcriptional activation. Furthermore, overexpression of CDK11(p58) decreased the stability of VDR through promoting its ubiquitin-proteasome-mediated degradation. Taken together, these results suggest that CDK11(p58) is involved in the negative regulation of VDR.
Mesh Terms:
Animals, COS Cells, Cercopithecus aethiops, Cyclin D3, Cyclins, Humans, Proteasome Endopeptidase Complex, Receptors, Calcitriol, Transcriptional Activation, Transfection, Ubiquitin, Ubiquitination
Animals, COS Cells, Cercopithecus aethiops, Cyclin D3, Cyclins, Humans, Proteasome Endopeptidase Complex, Receptors, Calcitriol, Transcriptional Activation, Transfection, Ubiquitin, Ubiquitination
Biochem. Biophys. Res. Commun.
Date: Aug. 28, 2009
PubMed ID: 19538938
View in: Pubmed Google Scholar
Download Curated Data For This Publication
126568
Switch View:
- Interactions 2
- PTM Genes 1