Reduced MAP kinase phosphatase-1 degradation after p42/p44MAPK-dependent phosphorylation.

The mitogen-activated protein (MAP) kinase cascade is inactivated at the level of MAP kinase by members of the MAP kinase phosphatase (MKP) family, including MKP-1. MKP-1 was a labile protein in CCL39 hamster fibroblasts; its degradation was attenuated by inhibitors of the ubiquitin-directed proteasome complex. MKP-1 was a target in ...
vivo and in vitro for p42(MAPK) or p44(MAPK), which phosphorylates MKP-1 on two carboxyl-terminal serine residues, Serine 359 and Serine 364. This phosphorylation did not modify MKP-1's intrinsic ability to dephosphorylate p44(MAPK) but led to stabilization of the protein. These results illustrate the importance of regulated protein degradation in the control of mitogenic signaling.
Mesh Terms:
Animals, Blood, Cell Cycle Proteins, Cell Division, Cell Line, Cricetinae, Culture Media, Cysteine Endopeptidases, Cysteine Proteinase Inhibitors, Dual Specificity Phosphatase 1, Estradiol, Humans, Immediate-Early Proteins, Leucine, Leupeptins, MAP Kinase Signaling System, Mitogen-Activated Protein Kinase 1, Mitogen-Activated Protein Kinase 3, Mitogen-Activated Protein Kinases, Multienzyme Complexes, Mutation, Nitrophenols, Organophosphorus Compounds, Phosphoprotein Phosphatases, Phosphorylation, Proteasome Endopeptidase Complex, Protein Phosphatase 1, Protein Tyrosine Phosphatases, Ubiquitins
Science
Date: Dec. 24, 1999
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