TORC2 signaling pathway guarantees genome stability in the face of DNA strand breaks.
A chemicogenetic screen was performed in budding yeast mutants that have a weakened replication stress response. This identified an inhibitor of target of rapamycin (TOR) complexes 1 and 2 that selectively enhances the sensitivity of sgs1Δ cells to hydroxyurea and camptothecin. More importantly, the inhibitor has strong synthetic lethality in ... combination with either the break-inducing antibiotic Zeocin or ionizing radiation, independent of the strain background. Lethality correlates with a rapid fragmentation of chromosomes that occurs only when TORC2, but not TORC1, is repressed. Genetic inhibition of Tor2 kinase, or its downstream effector kinases Ypk1/Ypk2, conferred similar synergistic effects in the presence of Zeocin. Given that Ypk1/Ypk2 controls the actin cytoskeleton, we tested the effects of actin modulators latrunculin A and jasplakinolide. These phenocopy TORC2 inhibition on Zeocin, although modulation of calcineurin-sensitive transcription does not. These results implicate TORC2-mediated actin filament regulation in the survival of low levels of DNA damage.
Mesh Terms:
Actins, Bicyclo Compounds, Heterocyclic, Bleomycin, Chromosomes, DNA Damage, DNA Replication, Genomic Instability, Glycogen Synthase Kinase 3, Multiprotein Complexes, Radiation, Ionizing, Saccharomyces cerevisiae, Saccharomyces cerevisiae Proteins, Signal Transduction, TOR Serine-Threonine Kinases, Thiazolidines, Transcription Factors
Actins, Bicyclo Compounds, Heterocyclic, Bleomycin, Chromosomes, DNA Damage, DNA Replication, Genomic Instability, Glycogen Synthase Kinase 3, Multiprotein Complexes, Radiation, Ionizing, Saccharomyces cerevisiae, Saccharomyces cerevisiae Proteins, Signal Transduction, TOR Serine-Threonine Kinases, Thiazolidines, Transcription Factors
Mol. Cell
Date: Sep. 26, 2013
PubMed ID: 24035500
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