Atypical recognition consensus of CIN85/SETA/Ruk SH3 domains revealed by target-assisted iterative screening.

Target-assisted iterative screening applied to random peptide libraries unveiled a novel and atypical recognition consensus shared by CIN85/SETA/Ruk SH3 domains, PX(P/A)XXR. Confirmed by mutagenesis and in vitro binding experiments, the novel consensus allowed for the accurate mapping of CIN85 SH3 binding sites within known CIN85 interactors, c-Cbl, BLNK, Cbl-b, AIP1/Alix, ...
SB1, and CD2 proteins, as well as the prediction of CIN85 novel-interacting partners in protein databases. Synaptojanin 1, PAK2, ZO-2, and TAFII70, which contain CIN85 SH3 recognition consensus sites, were selectively precipitated from mouse brain lysates by CIN85 SH3 domains in glutathione S-transferase pull-down experiments. A direct interaction of synaptojanin 1 and PAK2 with CIN85 SH3 domains was confirmed by Far Western blotting.
Mesh Terms:
Adaptor Proteins, Signal Transducing, Alanine, Amino Acid Sequence, Animals, Base Sequence, Binding Sites, Biochemistry, Blotting, Western, Brain, Carrier Proteins, Electrophoresis, Polyacrylamide Gel, Enzyme-Linked Immunosorbent Assay, Glutathione Transferase, Mice, Molecular Sequence Data, Mutagenesis, Mutagenesis, Site-Directed, Neoplasm Proteins, Nerve Tissue Proteins, Peptide Library, Phosphoric Monoester Hydrolases, Precipitin Tests, Protein Binding, Protein Structure, Tertiary, Recombinant Fusion Proteins, src Homology Domains
J. Biol. Chem.
Date: Sep. 05, 2003
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