BAIT

TRP53

Tp53, bbl, bfy, bhy, p44, p53, RP23-56I20.1
transformation related protein 53
GO Process (100)
GO Function (32)
GO Component (17)

Gene Ontology Biological Process

Mus musculus
PREY

REV3L

Rev, Rev3, Sez4
REV3-like, catalytic subunit of DNA polymerase zeta RAD54 like (S. cerevisiae)
GO Process (3)
GO Function (1)
GO Component (2)

Gene Ontology Molecular Function

Gene Ontology Cellular Component

Mus musculus

Phenotypic Enhancement

A genetic interaction is inferred when mutation or overexpression of one gene results in enhancement of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

siRNA screening identifies differences in the Fanconi anemia pathway in BALB/c-Trp53+/- with susceptibility versus C57BL/6-Trp53+/- mice with resistance to mammary tumors.

Boehringer M, Obermeier K, Griner N, Waldraff D, Dickinson E, Eirich K, Schindler D, Hagen M, Jerry DJ, Wiesmueller L

BALB/c mice heterozygous for Trp53 develop a high proportion of spontaneous mammary tumors, a phenotype distinct from other mouse strains. BALB/c-Trp53+/- female mice, thus, resemble the hereditary Li-Fraumeni syndrome (LFS) characterized by early-onset of breast cancer, even though LFS involves TP53 mutations, which may involve not only loss- but also gain-of-function. Previous analysis of tumors in BALB/c-Trp53+/- females showed frequent ... [more]

Oncogene Nov. 28, 2013; 32(48);5458-70 [Pubmed: 23435420]

Throughput

  • Low Throughput

Additional Notes

  • siRNA targeting the hit results in down-regulation of DNA repair in mice hemizygous for p53

Curated By

  • BioGRID