CG13432, CG13433, Dmel\CG13432, EP2356, EP2587, L(2)05510, l(2)k009920, lethal (2) 05510, Dmel_CG13432
GO Process (2)
GO Function (0)
GO Component (0)

Gene Ontology Biological Process

Drosophila melanogaster


CG10544, CG2509, Dmel\CG31547, Dmel_CG31547
CG31547 gene product from transcript CG31547-RB
GO Process (2)
GO Function (2)
GO Component (0)
Drosophila melanogaster

Phenotypic Suppression

A genetic interaction is inferred when mutation or over expression of one gene results in suppression of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.


Quasimodo mediates daily and acute light effects on Drosophila clock neuron excitability.

Buhl E, Bradlaugh A, Ogueta M, Chen KF, Stanewsky R, Hodge JJ

We have characterized a light-input pathway regulating Drosophila clock neuron excitability. The molecular clock drives rhythmic electrical excitability of clock neurons, and we show that the recently discovered light-input factor Quasimodo (Qsm) regulates this variation, presumably via an Na+, K+, Cl- cotransporter (NKCC) and the Shaw K+ channel (dKV3.1). Because of light-dependent degradation of the clock protein Timeless (Tim), constant ... [more]

Proc. Natl. Acad. Sci. U.S.A. Dec. 22, 2015; 113(47);13486-13491 [Pubmed: 27821737]


  • Low Throughput

Related interactions

InteractionExperimental Evidence CodeDatasetThroughputScoreCurated ByNotes
QSM CG31547

Bait protein expressed as a DNA binding domain (DBD) fusion and prey expressed as a transcriptional activation domain (TAD) fusion and interaction measured by reporter gene activation.


Curated By