BAIT
TOR1
SPBC30D10.10c
phosphatidylinositol kinase Tor1
GO Process (8)
GO Function (1)
GO Component (3)
Gene Ontology Biological Process
- TOR signaling [TAS]
- cell aging [TAS]
- cellular response to nitrogen starvation [IMP]
- cellular response to osmotic stress [IMP]
- phosphatidylinositol-mediated signaling [NAS]
- positive regulation of conjugation with cellular fusion [IMP]
- regulation of conjugation with cellular fusion [IMP]
- response to temperature stimulus [IMP]
Gene Ontology Molecular Function
Schizosaccharomyces pombe (972h)
PREY
TSC1
SPAC22F3.13
hamartin
GO Process (4)
GO Function (1)
GO Component (2)
Gene Ontology Biological Process
Gene Ontology Molecular Function
Gene Ontology Cellular Component
Schizosaccharomyces pombe (972h)
Synthetic Lethality
A genetic interaction is inferred when mutations or deletions in separate genes, each of which alone causes a minimal phenotype, result in lethality when combined in the same cell under a given condition.
Publication
Opposite effects of tor1 and tor2 on nitrogen starvation responses in fission yeast.
The TOR protein kinases exhibit a conserved role in regulating cellular growth and proliferation. In the fission yeast two TOR homologs are present. tor1(+) is required for starvation and stress responses, while tor2(+) is essential. We report here that Tor2 depleted cells show a phenotype very similar to that of wild-type cells starved for nitrogen, including arrest at the G(1) ... [more]
Genetics Mar. 01, 2007; 175(3);1153-62 [Pubmed: 17179073]
Throughput
- Low Throughput
Ontology Terms
- phenotype: inviable (APO:0000112)
Curated By
- BioGRID