BAIT

KRAS

AI929937, K-ras, Ki-ras, Kras-2, Kras2, p21B, ras, RP24-359O2.1
v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog
Mus musculus
PREY

ATM

AI256621, C030026E19Rik
ataxia telangiectasia mutated
GO Process (35)
GO Function (9)
GO Component (5)
Mus musculus

Positive Genetic

Mutations/deletions in separate genes, each of which alone causes a minimal phenotype, but when combined in the same cell results in a less severe fitness defect than expected under a given condition. This term is reserved for high or low throughput studies with scores.

Publication

Genome-wide CRISPR Screen to Identify Genes that Suppress Transformation in the Presence of Endogenous KrasG12D.

Huang J, Chen M, Xu ES, Luo L, Ma Y, Huang W, Floyd W, Klann TS, Kim SY, Gersbach CA, Cardona DM, Kirsch DG

Cooperating gene mutations are typically required to transform normal cells enabling growth in soft agar or in immunodeficient mice. For example, mutations in Kras and transformation-related protein 53 (Trp53) are known to transform a variety of mesenchymal and epithelial cells in vitro and in vivo. Identifying other genes that can cooperate with oncogenic Kras and substitute for Trp53 mutation has ... [more]

Sci Rep Dec. 20, 2018; 9(1);17220 [Pubmed: 31748650]

Throughput

  • High Throughput

Additional Notes

  • CRISPR GI screen
  • Cell Line: MEF
  • Experimental Setup: non-immortal cell line grown on soft agar or in mouse models to look for co-mutations that lead to cell transformation (positive selection)
  • GIST: A-phenotypic positive genetic interaction
  • Library: Selected targets from Brie and mGECKO
  • Significance Threshold: NA

Curated By

  • BioGRID