HAC1
Gene Ontology Biological Process
- endoplasmic reticulum unfolded protein response [IMP]
- negative regulation of transcription from RNA polymerase II promoter during meiosis [IDA, IMP]
- positive regulation of transcription from RNA polymerase II promoter [IDA, IMP]
- positive regulation of transcription from RNA polymerase II promoter involved in unfolded protein response [IMP]
Gene Ontology Molecular Function
SOD1
Gene Ontology Biological Process
- age-dependent response to reactive oxygen species involved in chronological cell aging [IMP]
- cellular copper ion homeostasis [IMP]
- cellular zinc ion homeostasis [IMP]
- fungal-type cell wall organization [IMP]
- negative regulation of cellular respiration [IMP]
- positive regulation of sequence-specific DNA binding transcription factor activity [IMP]
- positive regulation of transcription from RNA polymerase II promoter in response to oxidative stress [IMP]
- protein stabilization [IMP, IPI]
- removal of superoxide radicals [IBA]
- superoxide metabolic process [IMP]
Gene Ontology Molecular Function
Gene Ontology Cellular Component
Synthetic Lethality
A genetic interaction is inferred when mutations or deletions in separate genes, each of which alone causes a minimal phenotype, result in lethality when combined in the same cell under a given condition.
Publication
Cu, Zn superoxide dismutase and NADP(H) homeostasis are required for tolerance of endoplasmic reticulum stress in Saccharomyces cerevisiae.
Genome-wide screening for sensitivity to chronic endoplasmic reticulum (ER) stress induced by dithiothreitol and tunicamycin (TM) identified mutants deleted for Cu, Zn superoxide dismutase (SOD) function (SOD1, CCS1) or affected in NADPH generation via the pentose phosphate pathway (TKL1, RPE1). TM-induced ER stress led to an increase in cellular superoxide accumulation and an increase in SOD1 expression and Sod1p activity. ... [more]
Throughput
- Low Throughput
Ontology Terms
- phenotype: metal resistance (APO:0000090)
- phenotype: inviable (APO:0000112)
Additional Notes
- DTT
- copper
- paraquat
- tunicamycin
Related interactions
Interaction | Experimental Evidence Code | Dataset | Throughput | Score | Curated By | Notes |
---|---|---|---|---|---|---|
SOD1 HAC1 | Phenotypic Enhancement Phenotypic Enhancement A genetic interaction is inferred when mutation or overexpression of one gene results in enhancement of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene. | Low | - | BioGRID | 349103 | |
HAC1 SOD1 | Synthetic Growth Defect Synthetic Growth Defect A genetic interaction is inferred when mutations in separate genes, each of which alone causes a minimal phenotype, result in a significant growth defect under a given condition when combined in the same cell. | Low | - | BioGRID | 349100 |
Curated By
- BioGRID