BAIT

WNT11

wingless-type MMTV integration site family, member 11
GO Process (35)
GO Function (6)
GO Component (4)
Mus musculus
PREY

TCF4

5730422P05Rik, ASP-I2, E2-2, E2.2, ITF-2, ITF-2b, ITF2, ME2, MITF-2A, MITF-2B, SEF-2, SEF2, SEF2-1, TFE, Tcf-4, bHLHb19
transcription factor 4
Mus musculus

Phenotypic Suppression

A genetic interaction is inferred when mutation or over expression of one gene results in suppression of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

Wnt11 promotes osteoblast maturation and mineralization through R-spondin 2.

Friedman MS, Oyserman SM, Hankenson KD

Wnt11 signals through both canonical (beta-catenin) and non-canonical pathways and is up-regulated during osteoblast differentiation and fracture healing. In these studies, we evaluated the role of Wnt11 during osteoblastogenesis. Wnt11 overexpression in MC3T3E1 pre-osteoblasts increases beta-catenin accumulation and promotes bone morphogenetic protein (BMP)-induced expression of alkaline phosphatase and mineralization. Wnt11 dramatically increases expression of the osteoblast-associated genes Dmp1 (dentin matrix ... [more]

J. Biol. Chem. May. 22, 2009; 284(21);14117-25 [Pubmed: 19213727]

Throughput

  • Low Throughput

Ontology Terms

  • phenotype: abnormal bone mineralization (MP:0002896)
  • phenotype: abnormal protein level (MP:0008469)

Additional Notes

  • expression of a dominant negative tcf4 blocks the effects of wnt11 overexpression, including expression of specific genes and mineralization

Curated By

  • BioGRID