PREY

CTNNB1

Bfc, Catnb, Mesc
catenin (cadherin associated protein), beta 1
GO Process (137)
GO Function (14)
GO Component (29)

Gene Ontology Biological Process

Mus musculus

Phenotypic Enhancement

A genetic interaction is inferred when mutation or overexpression of one gene results in enhancement of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

Wnt9a signaling is required for joint integrity and regulation of Ihh during chondrogenesis.

Spaeter D, Hill TP, O'sullivan RJ, Gruber M, Conner DA, Hartmann C

Joints, which separate skeleton elements, serve as important signaling centers that regulate the growth of adjacent cartilage elements by controlling proliferation and maturation of chondrocytes. Accurate chondrocyte maturation is crucial for endochondral ossification and for the ultimate size of skeletal elements, as premature or delayed maturation results predominantly in shortened elements. Wnt9a has previously been implicated as being a player ... [more]

Development Aug. 01, 2006; 133(15);3039-49 [Pubmed: 16818445]

Throughput

  • Low Throughput

Ontology Terms

  • abnormal chondrocyte morphology (MP:0000166)

Additional Notes

  • mutation of beta-catenin further decreases hedgehog expression in a Wnt9a mutant

Curated By

  • BioGRID