PREY

SP1

Sp1 transcription factor
Homo sapiens

Phenotypic Enhancement

A genetic interaction is inferred when mutation or overexpression of one gene results in enhancement of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

Homocysteine upregulates soluble epoxide hydrolase in vascular endothelium in vitro and in vivo.

Zhang D, Xie X, Chen Y, Hammock BD, Kong W, Zhu Y

Hyperhomocysteinemia is a risk factor of atherogenesis. Soluble epoxide hydrolase (sEH) is a major enzyme that hydrolyzes epoxyeicosatrienoic acids and attenuates their cardiovascular protective effects. Whether homocysteine (Hcy) regulates sEH and the underlying mechanism remains elusive.To elucidate the mechanism by which Hcy regulates sEH expression and endothelial activation in vitro and in vivo.Hcy treatment in cultured human endothelial cells dose-dependently ... [more]

Circ. Res. Mar. 16, 2012; 110(6);808-17 [Pubmed: 22354938]

Throughput

  • Low Throughput

Additional Notes

  • figure S4. ATF6 and SP1 were co-contributed to Hcy-induced sEH expression

Curated By

  • BioGRID