BAIT

NFE2L2

AI194320, Nrf2, RP23-374O4.2
nuclear factor, erythroid derived 2, like 2
GO Process (24)
GO Function (10)
GO Component (7)
Mus musculus
PREY

ATG7

1810013K23Rik, Agp7, Apg7l, Atg7l, Gm21553
autophagy related 7
GO Process (37)
GO Function (5)
GO Component (4)

Gene Ontology Cellular Component

Mus musculus

Phenotypic Suppression

A genetic interaction is inferred when mutation or over expression of one gene results in suppression of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1.

Komatsu M, Kurokawa H, Waguri S, Taguchi K, Kobayashi A, Ichimura Y, Sou YS, Ueno I, Sakamoto A, Tong KI, Kim M, Nishito Y, Iemura S, Natsume T, Ueno T, Kominami E, Motohashi H, Tanaka K, Yamamoto M

Impaired selective turnover of p62 by autophagy causes severe liver injury accompanied by the formation of p62-positive inclusions and upregulation of detoxifying enzymes. These phenotypes correspond closely to the pathological conditions seen in human liver diseases, including alcoholic hepatitis and hepatocellular carcinoma. However, the molecular mechanisms and pathophysiological processes in these events are still unknown. Here we report the identification ... [more]

Nat. Cell Biol. Mar. 01, 2010; 12(3);213-23 [Pubmed: 20173742]

Throughput

  • Low Throughput

Ontology Terms

  • phenotype: abnormal liver morphology (MP:0000598) [abnormal liver morphology (MP:0000598)]

Additional Notes

  • figure 5. Liver injury in autophagy-deficient mice (atg7-/-) is alleviated by loss of Nrf2.

Curated By

  • BioGRID