BAIT

SNCA

NACP, PARK1, PARK4, PD1
synuclein, alpha (non A4 component of amyloid precursor)
GO Process (45)
GO Function (20)
GO Component (16)

Gene Ontology Biological Process

Homo sapiens
PREY

DNAJC5

2610314I24Rik, AU018536, Csp, RP23-33L3.3
DnaJ (Hsp40) homolog, subfamily C, member 5
GO Process (1)
GO Function (2)
GO Component (7)
Mus musculus

Phenotypic Suppression

A genetic interaction is inferred when mutation or over expression of one gene results in suppression of any phenotype (other than lethality/growth defect) associated with mutation or over expression of another gene.

Publication

Alpha-synuclein cooperates with CSPalpha in preventing neurodegeneration.

Chandra S, Gallardo G, Fernandez-Chacon R, Schlueter OM, Suedhof TC

Alpha-synuclein and cysteine-string protein-alpha (CSPalpha) are abundant synaptic vesicle proteins independently linked to neurodegeneration. Dominantly inherited mutations in alpha-synuclein cause Parkinson's disease, but the physiological role of alpha-synuclein remains unknown. Deletion of CSPalpha produces rapidly progressive neurodegeneration in mice, presumably because the cochaperone function of CSPalpha is essential for neuronal survival. Here, we report the surprising finding that transgenic expression ... [more]

Cell Nov. 04, 2005; 123(3);383-96 [Pubmed: 16269331]

Throughput

  • Low Throughput

Additional Notes

  • figure 3. Transgenic human SNCA Reverses the Neuropathology Induced by Deletion of Dnajc5.

Curated By

  • BioGRID