BAIT

PTEN

2310035O07Rik, A130070J02Rik, AI463227, B430203M17Rik, MMAC1, TEP1
phosphatase and tensin homolog
GO Process (72)
GO Function (13)
GO Component (13)

Gene Ontology Biological Process

Mus musculus
PREY

FBXW7

1110001A17Rik, AGO, Cdc4, Fbw7, Fbwd6, Fbx30, Fbxo30, Fbxw6, SEL-10
F-box and WD-40 domain protein 7
GO Process (18)
GO Function (7)
GO Component (10)
Mus musculus

Synthetic Haploinsufficiency

A genetic interaction is inferred when mutations or deletions in separate genes, at least one of which is hemizygous, cause a minimal phenotype alone but result in lethality when combined in the same cell under a given condition.

Publication

Pten regulates Aurora-A and cooperates with Fbxw7 in modulating radiation-induced tumor development.

Kwon YW, Kim IJ, Wu D, Lu J, Stock WA, Liu Y, Huang Y, Kang HC, Delrosario R, Jen KY, Perez-Losada J, Wei G, Balmain A, Mao JH

The Aurora-A kinase gene is frequently amplified and/or over-expressed in a variety of human cancers, leading to major efforts to develop therapeutic agents targeting this pathway. Here we demonstrate that Aurora-A is targeted for ubiquitination and subsequent degradation by the F-box protein FBXW7 in a process that is regulated by GSK3β. Using a series of truncated Aurora-A proteins and site ... [more]

Unknown Apr. 18, 2012; 0(0); [Pubmed: 22513362]

Throughput

  • Low Throughput

Additional Notes

  • figure 7. Pten interacts with Fbxw7 in suppressing radiation-induced tumorigenesis.

Curated By

  • BioGRID